Cellular Glucose & Tumor Cachexia Simulator

Interactive systemic metabolic flux modeling: why zero dietary sugar fails to starve tumor cells and drives skeletal muscle breakdown (cachexia).

Systemic Glucose & Organ Transporter Network Day 14.0 / 14
Dietary Glucose
Gluconeogenic Glucose
Muscle Amino Acids
Tumor GLUT1 Flux
Sub-cellular Transporter Mechanics (Warburg Effect)

Tumor (GLUT1): High affinity, insulin-independent expression. Uptake rate remains saturated even at baseline fasting glucose (~70 mg/dL).
Muscle (GLUT4): Insulin-dependent, down-regulated during low dietary intake to preserve plasma glucose for brain and tumor uptake.

Intervention & Telemetry Dashboard
Plasma Blood Glucose
92 mg/dL
Homeostasis Maintained
Liver Gluconeogenesis
12 g/day
Baseline hepatic rate
Muscle Catabolism
15 g AA/day
Normal protein turnover
Tumor GLUT1 Glucose Uptake
48 g/day
Continuous constitutive uptake
Systemic Outcome & Cachexia Risk: Normal (12%)
At 150g/day sugar intake, dietary glucose fulfills CNS requirement (~120g/day) without severe skeletal muscle breakdown.