Skeletal Muscle Metabolic Flux Engine

Simulating 12S rRNA peptide export, folate cycle feedback, AICAR buildup & AMPK Thr172 phosphorylation

Normal Metabolism
Endogenous: ~5-12 nM | High Supp: >50 nM
Alters basal folate clearance and GLUT4 reserve
Mitochondrial Origin: 16-aa MOTS-c (mtDNA 12S rRNA)
Inhibition: 10-formyl-THF Folate Cycle & Purine synthesis
Activation: AICAR accumulation & AMPK phosphorylation
Intracellular AICAR
3.12 µM
10-formyl-THF Flux
48.2%
AMPK Activation
2.41x
GLUT4 Translocation
+64%

Clinical Workspace & Regulatory Evidence

NCT07505745 human Phase 2a parameters, Matsuda Index projections, & PCAC vote context

Hudson Biotech / PKU Shenzhen Hospital (Phase 2a) Recruiting Since Apr 1, 2026
Study Identifier NCT07505745 (Double-blind RCT)
Dosing Architecture Once-daily Subcutaneous (12 Wks)
Masking Level Quadruple (Participant, Care, Inv, Assessor)
Primary Readout Matsuda Index (OGTT, Feb 2027)
Simulated 12-Week Matsuda Index Shift (Baseline vs Modeled End-of-Study) +42% Projected
Analog Historical Control: CB4211, an engineered synthetic analogue developed by CohBar, underwent Phase 1a/1b testing in non-alcoholic fatty liver disease (NAFLD) in 2021 before termination. MOTS-MET represents the first registered trial of native, sequence-identical mitochondrial MOTS-c.
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