Rb Dual-Role Transcription & Tumor Suppression Simulator

Mechanistic kinetic model of canonical G1/S arrest versus paradoxical co-activator flux

Molecular Regulatory Circuit (D3.js) Paradoxical Transcription
Canonical G1 Arrest 28.4%
Oncogenic Rb Flux 0.68
Net Suppression Score -0.12
Phospho-Rb Ratio 0.72
Phenotype Phase-Plane & Binding Curves
Active Pathway Regime: Paradoxical Rb-Driven Transcriptional Activation. High chromatin co-factor recruitment overrides unphosphorylated Rb's classical cell cycle arrest.
Experimental Perturbations
0.72
0.85
1.00
0.00 µM
Gene Target Promoter Occupancy
E2F Target (Cyclin E1/A2, Proliferation) 43.6%
Rb-Coactivated Loci (Survival/Metabolism) 68.0%
E2F-Rb Repressive Complex Density 28.4%
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