HGPS Progeria Cellular Dynamics

LMNA Mutation Biophysics
Real-time Nuclear Envelope Mechanics & Chromatin Architecture
Normal Lamin A/C
Progerin Accumulation
DNA Damage Focus
Drag nuclear membrane nodes to test structural stiffness
Cell Line / Type
Progerin Level 0.85
Mechanical Stress 0.50
FTI Dosage (Lonafarnib) 0 mg
LMNA Mutation Rate 1.00
Nuclear Morphometry PATHOLOGY
Circularity Index 0.42
Blebbing Frequency 0.78
DNA Damage Foci Count 18
FTI Rescue Efficiency 0%
Senescence Onset Day 14
Molecular Mechanism
Classic HGPS is driven by the c.1824C>T mutation in the LMNA gene, activating a cryptic splice site. This produces truncated Progerin which retains a toxic farnesyl lipid anchor, permanently embedding into the nuclear lamina and destabilizing membrane mechanics.
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CIRCULARITY: 0.42 | BLEBBING: 0.78 | FOCI: 18 | RESCUE: 0%
Vascular & Cellular Aging Trajectory Comparison Age Equivalent: 68 Biological Years
0 yrs (Birth) 5 yrs (Early Stiffness) 12 yrs (Cardiovascular Calcification) 20+ yrs (Advanced Rescue/Longevity Target)
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